A fascinating article recently appeared in the Guardian, about a woman who found a way to overcome a very particular type of learning disability and has apparently helped a great many children since.
Articles
Introduction
In 1997, the U.S. Congress asked the Director of the National Institute of Child Health and Human Development (NICHD) at the National Institutes of Health, in consultation with the Secretary of Education, to convene a national panel to assess the effectiveness of different approaches used to teach children to read. For over two years, the National Reading Panel reviewed research-based knowledge on reading instruction and held open panel meetings in Washington, DC, and regional meetings across the United States.
"Consolidation" is a term that is bandied about a lot in recent memory research. Here's my take on what it means.
Becoming a memory
Initially, information is thought to be encoded as patterns of neural activity — cells "talking" to each other. Later, the information is coded in more persistent molecular or structural formats (e.g., the formation of new synapses). It has been assumed that once this occurs, the memory is "fixed" — a permanent, unchanging, representation.
In a recent news report, I talked about how walking through doorways creates event boundaries, requiring us to update our awareness of current events and making information about the previous location less available. I commented that we should be aware of the consequences of event boundaries for our memory, and how these contextual factors are important elements of our filing system. I want to talk a bit more about that.
People are poor at assessing their own memory
One thing research seems to show rather consistently is that, for older adults in particular, beliefs about one's own memory performance have little to do with one's actual memory performance¹. People who believe they have a poor memory are usually no worse at remembering than those who believe they have a good memory.
LBD: What is it?
Lewy Body Dementia is so called because the brains of affected people develop abnormal spherical masses of protein, called Lewy bodies, inside nerve cells. Lewy bodies are associated with Parkinson’s disease as well as dementia. Thus Lewy body dementia can refer to both Parkinson’s disease dementia and “dementia with Lewy bodies”. Lewy bodies are also often found in the brains of those with Alzheimer’s disease.
Unlike Alzheimer’s, however, dementia with Lewy bodies characteristically (but not invariably) begins with visual hallucinations.
What is it?
Frontotemporal dementia is a disorder of the frontal lobes and includes what was known as primary progressive aphasia. Although it occurs far less often than Alzheimer's disease, among dementia sufferers younger than 65 it is estimated to occur at about the same rate. In other words, frontotemporal dementia is, unlike the most common dementias, not a disorder of age. Most sufferers become symptomatic in their 50s and 60s.
When we are presented with new information, we try and connect it to information we already hold. This is automatic. Sometimes the information fits in easily; other times the fit is more difficult — perhaps because some of our old information is wrong, or perhaps because we lack some of the knowledge we need to fit them together.
I talk a lot about how working memory constrains what we can process and remember, but there’s another side to this — long-term memory acts on working memory. That is, indeed, the best way of ‘improving’ your working memory — by organizing and strengthening your long-term memory codes in such a way that large networks of relevant material are readily accessible.
Oddly enough, one of the best ways of watching the effect of long-term memory on working memory is through perception.